[Product name] Flaxseed extract, Flaxseed P.E., Flax-seed extract, Flaxseed SDG
[Latin Name] Linum usitatissimum L
[Plant Source] Flaxseed harvested in Inner Mongolia
[Specifications] lignans (SDG) 20%,40%,70%,80%
[Apprearance] Yellowish Powder
[Particle size] 80 Mesh
[Loss on drying] ≤ 5.0%
[Heavy Metal] ≤10PPM
[Extract solvents] Ethanol
[GMO Statement] GMO Free
[KOSHER] Yes
[Microbe] Total Aerobic Plate Count: ≤1000CFU/G
Yeast & Mold: ≤100 CFU/G
[Characteristics]
Flax seed is an ancient grain that has been used in the diets of humans for thousands of years. Hippocrates, the father of medicine, used flax seed for the relief of intestinal discomfort in 650 BC.
The seeds and oil of the flax plant contain substances which promote good health. Flaxseed and flaxseed oil are rich in alpha-linolenic acid (ALA), an essential fatty acid that appears to be beneficial for heart disease. ALA belongs to a group of substances called omega-3 fatty acids.
SDG is classified as a phytoestrogen since it is a plant-derived, nonsteroid compound that possesses estrogen-like activity. SDG has weak estrogenic activity. The level of SDG in flaxseed typically varies between 0.6% and 1.8%. Lignans are one of the two major classes of phytoestrogens; The other class is the isoflavones. Plant lignans are polyphenolic substances derived from phenylalanine via dimerization of substituted cinnamic alcohols. Mammalian lignans are lignans derived from plant lignans. For example, following ingestion, SDG is converted to the aglycone secoisolariciresinol, which is then metabolized to the mammalian lignans enterolactone and enterodiol. Most of the effects of oral SDG are mediated by enterolactone and enterodiol.
[Storage] Store in cool & dry area, keep away from the direct light and heat.
[Shelf life]24 Months
[Package] Packed in paper-drums and two plastic-bags inside. Net weight:25kgs/drum.
[Function]
Improve estrogenic and antioxidant activities, increased anticarcinogenic, antiatherogenic and antidiabetic activities, reducing hypercholesterolemic atherosclerosis oxidative stress and lowering serum levels of cholesterol and low-density lipoprotein-cholesterol and raising serum levels of high-density lipoprotein-cholesterol.